Jump to content
IGNORED

Chernobyl - HBO (2019)


eumeswil

Recommended Posts

dobila sam link za konferenciju, pa ostavljam ovde ako nekog zanima 

registracija je besplatna

 

Директорат за радијациону и нуклеарну сигурност и безбедност Србије (СРБАТОМ) 26. априла организује онлајн конференцију „Нуклеарна сигурност данас: Чернобиљ 35 година после“, a поводом тридесетпетогодишњице од нуклеарног акцидента у Чернобиљу, једног од најзначајнијих акцидената у историји човечанства који је значајно утицао на нуклеарну енергетику и сигурност у свету.

Конференцију ће отворити директор СРБАТОМ-а Слађан Велинов и амбасадор Украјине у Републици Србији Њ. Е. Олександр Александрович.

Циљ догађаја је упознавање шире јавности са самим акцидентом, а говориће се и о ублажавању његових последица, декомисији нуклеарне електране, санацији и ремедијацији локације као и о реакцијама у Србији и свету на овај нуклеарни акцидент. Цео програм конференције, можете погледати на линку.

Конференцију можете пратити на YouTube каналу СРБАТОМ-а, а за праћење се можете регистровати путем линка.

Надамо се да ћете препознати значај догађаја и одазвати се нашем позиву. Уколико сматрате да ће ова конференција још некоме значити, молимо вас проследите му овај позив.

Link to comment
8 minutes ago, beyoncé said:

dobila sam link za konferenciju, pa ostavljam ovde ako nekog zanima 

registracija je besplatna

Da me ne shvatis pogresno, tema je u svakom slucaju interesantna i vazna.

Ali, samo iz uvida u nesto materijala i razgovora sa par poznanika, vise sam nego siguran da organizatori, sa sve njegovom ekselencijom nikako nisu firme od poverenja, a jos sam sigurniji u kom prevcu ce prica da bude tendenciozno gurana i odgurana...

Kvalitetnija prica, obelezavanje Cernobila, dobila bi se ovde na forumu, tipujem na nevidjeno... :) 

Link to comment

U nedostatku nove sezone, malo nauke:
 

Quote

 

Chernobyl radiation damage 'not passed to children'

 

There is no "additional DNA damage" in children born to parents who were exposed to radiation from the Chernobyl explosion before they were conceived.

 

This is according to the first study to screen the genes of children whose parents were enlisted to help in the clean-up after the nuclear accident.

 

 

link

  • +1 2
Link to comment
On 24.4.2021. at 23:49, Frank Pembleton said:

@pt 2.0 svrati na temu...

j'y suis j'y reste :fantom:

 

ako treba da prokomentarišem ovo iznad, lepo je što su studijom potvrdili ono što se tvrdi all along: nema direktnih dokaza da izlaganje roditelja zračenju dovodi do razvoja naslednih bolesti u potomstvu.

to sam slušala na predavanjima gospodina Abel Julio Gonzáleza, to tvrdi i globalna biblija za oblast zaštite od zračenja, ICRP Publication 103, "The 2007 Recommendations of the International Commission on Radiological Protection", 3.2.2 Risk of heritable effects:

"There continues to be no direct evidence that exposure of parents to radiation leads to excess heritable disease in offspring. However, the Commission judges that there is compelling evidence that radiation causes heritable effects in experimental animals. Therefore, the Commission prudently continues to include the risk of heritable effects in its system of radiological protection."

i dalje, u istom poglavlju: "The Commission also notes reports (reviewed in UNSCEAR, 2001) which argue, on the basis of A-bomb survivor and mouse genetic data, that the risk of heritable diseases tended to be overestimated in the past."

 

celo poglavlje dajem u spojliši.

Spoiler

 

3.2.2. Risk of heritable effects

(74) There continues to be no direct evidence that exposure of parents to radiation leads to excess heritable disease in offspring. However, the Commission judges that there is compelling evidence that radiation causes heritable effects in experimental animals. Therefore, the Commission prudently continues to include the risk of heritable effects in its system of radiological protection. Table 1. Detriment-adjusted nominal risk coefficients (102 Sv1 ) for stochastic effects after exposure to radiation at low dose rate. Exposed population Cancer Heritable effects Total Present1 Publ. 60 Present1 Publ. 60 Present1 Publ. 60 Whole 5.5 6.0 0.2 1.3 5.7 7.3 Adult 4.1 4.8 0.1 0.8 4.2 5.6 1 Values from Annex A. ICRP Publication 103 53

(75) The Commission also notes reports (reviewed in UNSCEAR, 2001) which argue, on the basis of A-bomb survivor and mouse genetic data, that the risk of heritable diseases tended to be overestimated in the past. There are some post-1990 human and animal data on the quantitative aspects of radiation-induced germ cell mutation that impact on the Commission’s judgement on the risk of induction of genetic disease expressing in future generations. There have also been substantial advances in the fundamental understanding of human genetic diseases and the process of germ line mutagenesis including that occurring after radiation. The Commission has reappraised the methodology used in Publication 60 for the estimation of heritable risks including risks of multifactorial diseases (Publication 83, ICRP, 1999b).

(76) The Commission has now adopted a new framework for the estimation of heritable risks that employs data from human and mouse studies (UNSCEAR, 2001, NAS/NRC, 2006). Also, for the first time, a scientifically justified method for the estimation of risk of multifactorial disease has been included. Mouse studies continue to be used to estimate genetic risks because of the lack of clear evidence in humans that germline mutations caused by radiation result in demonstrable genetic effects in offspring.

(77) The new approach to heritable risks continues to be based on the concept of the doubling dose (DD) for disease-associated mutations used in Publication 60. However, the methodology differs in that recoverability of mutations in live births is allowed for in the estimation of DD. An additional difference is that direct data on spontaneous human mutation rates are used in conjunction with radiationinduced mutation rates derived from mouse studies. This new methodology (see Annex A, Box A.2) is based on the UNSCEAR 2001 report and has also been used recently by NAS/NRC (2006). In Publication 60 genetic risks were expressed at a theoretical equilibrium between mutation and selection. In the light of further knowledge the Commission judges that many of the underlying assumptions in such calculations are no longer sustainable. The same view has been expressed by UNSCEAR (2001) and NAS/NRC (2006). Accordingly, the Commission now expresses genetic risks up to the second generation only.

(78) The Commission judges that this procedure will not lead to a significant underestimation of heritable effects. This issue is discussed in UNSCEAR (2001) and in detail in Annex A where it is argued that there are no substantial differences between genetic risks expressed at 2 and 10 generations.

(79) The Commission’s present estimate of genetic risks up to the second generation of about 0.2% per Gy is essentially the same as that cited by UNSCEAR (2001) (see Annex A and UNSCEAR 2001, Table 46). However, given the major changes in methodology, the close similarity of the present second generation risk to that of Publication 60 is coincidental. The present value relates to continuous low-dose-rate exposures over these two generations.

 

 

Link to comment

Join the conversation

You can post now and register later. If you have an account, sign in now to post with your account.

Guest
Reply to this topic...

×   Pasted as rich text.   Paste as plain text instead

  Only 75 emoji are allowed.

×   Your link has been automatically embedded.   Display as a link instead

×   Your previous content has been restored.   Clear editor

×   You cannot paste images directly. Upload or insert images from URL.

×
×
  • Create New...